Rainbow Herbicides

The Rainbow Herbicides are a group of "tactical use" chemicals used by the United States military in Southeast Asia during the Vietnam War. Success with Project AGILE field tests with herbicides in South Vietnam in 1961 and inspiration by the British use of herbicides and defoliants during the Malayan Emergency in the 1950s led to the formal herbicidal program Trail Dust (see Operation Ranch Hand). Herbicidal warfare is the use of substances primarily designed to destroy the plant-based ecosystem of an agricultural food production and/or to destroy foliage which provides the enemy cover.

Four USAF C-123s spraying Rainbow Herbicide over South Vietnam as part of Operation Ranch Hand
Agent Orange stored at Johnston Atoll in 1976, following the end of US involvement in Vietnam

Background

The United States discovered 2,4-dichlorophenoxyacetic acid (2,4-D) during World War II. It was recognized as toxic and was combined with large amounts of water or oil to function as a weed-killer. Army experiments with the chemical eventually led to the discovery that 2,4-D combined with 2,4,5-trichlorophenoxyacetic acid (2,4,5-T) yielded a more potent herbicide.[1] It was found that 2,4,5-T was contaminated with 2,3,7,8-tetrachlorodibenzodioxin (TCDD), commonly called "dioxin". Researcher Alvin Lee Young found that samples of 2,4,5-T in Agents Pink and Green had doubled the TCDD concentration of Agents Purple or Orange.[2]

Types

Of the agents used in southeast Asia, their active ingredients and years used were as follow:[3]

Use
Professor Nguyen Thi Ngoc Phuong, at Từ Dũ Obstetrics and Gynecology Hospital is pictured with a group of handicapped children, most of them victims of Agent Orange.

In Vietnam, the early large-scale defoliation missions (1962-1964) used 8,208 gallons of Agent Green, 122,792 gallons of Pink, and 145,000 of Purple. These were dwarfed by the 11,712,860 gallons of Orange (both versions) used from 1965 to 1970. Agent White started to replace Orange in 1966; 5,239,853 gallons of White were used. The only agent used on a large scale in an anti-crop role was Blue, with 2,166,656 gallons used.[10] The bombardment occurred most heavily in the area of the Ho Chi Minh Trail.[1]

The rainbow herbicides damaged the ecosystems and cultivated lands of Vietnam, and led to buildup of dioxins in the regional food chain.[1] About 4.8 million people were affected.[11]

In addition to testing and using the herbicides in Vietnam, Laos and Cambodia, the US military also tested the "Rainbow Herbicides" and many other chemical defoliants and herbicides in the US,[12] Canada, Puerto Rico, Korea, India, and Thailand[13] from the mid-1940s to the late 1960s. Herbicide persistence studies of Agents Orange and White were conducted in the Philippines.[14] The Philippine herbicide test program which was conducted in cooperation with the University of the Philippines, College of Forestry and was also described in a 1969 issue of The Philippine Collegian, the college's newspaper. Super or enhanced Agent Orange was tested by representatives from Fort Detrick and Dow chemical in Texas, Puerto Rico, and Hawaii and later in Malaysia in a cooperative project with the International Rubber Research Institute.[8] Picloram in Agent White and Super-Orange was contaminated by hexachlorobenzene (HCB) a dioxin-like carcinogen. The Canadian government also tested these herbicides and used them to clear vegetation for artillery training.[15]

A 2003 study in Nature found that the military underreported its use of rainbow herbicides by 2,493,792 U.S. gallons.[11]

Long-term effects

Vietnam remains heavily contaminated by dioxin-like compounds, which are classified as persistent organic pollutants. These compounds remain in the water table and have built up in the tissues of local fauna. However, the contamination has begun to deteriorate, and the forest canopy has regrown somewhat since the Vietnam War.[1]

Dioxins are endocrine disruptors and may have effects on the children of people who were exposed.[1]

Rainbow herbicides and other dioxin-like compounds, are endocrine disruptors, and evidence suggests that they continue to have long-term health consequences many years after exposure. Because they mimic, or interfere with, hormonal function, adverse effects can include problems with reproduction, growth and development, immune function, and metabolic function. As an example, dioxins and dioxin-like compounds influence the hormone dehydoepiandosterone (DHEA), which has a role in the determination of male or female sex characteristics. There have been thousands of documented instances of health problems and birth defects associated with rainbow herbicide exposure in Vietnam, where tested levels remain high in the soil, water, and atmosphere, decades after initial exposure.

Soldiers exposed to Rainbow Herbicides in Southeast Asia reported long-term health effects, which led to several lawsuits against the U.S. government and the manufacturers of the chemical.[1]

See also

References
  1. Ornitz, Sheri L. "Agent Orange and its Continuing Effects". British Travel Health Association Journal, vol. 10. Winter 2007.
  2. Alvin L. Young (2009-04-21). The History, Use, Disposition and Environmental Fate of Agent Orange. Springer. p. 34. ISBN 978-0-387-87486-9.
  3. Stellman, Jeanne; Stellman, Steven D.; Christian, Richard; Weber, Tracy; Tomasallo, Carrie (17 April 2003). "The extent and patterns of usage of Agent Orange and other herbicides in Vietnam" (PDF). Nature. 422 (6933): 681–7. Bibcode:2003Natur.422..681S. doi:10.1038/nature01537. ISSN 0028-0836. PMID 12700752. S2CID 4419223.
  4. Committee to Review the Health Effects in Vietnam Veterans of Exposure to Herbicides; Institute of Medicine (1994). Veterans and Agent Orange: Health Effects of Herbicides Used in Vietnam. National Academies Press. pp. 89–90. ISBN 978-0-309-55619-4.
  5. Stephen Bull (2004). Encyclopedia of Military Technology and Innovation. Greenwood Publishing Group. p. 6. ISBN 978-1-57356-557-8.
  6. Daniel Vallero (2011). Biomedical Ethics for Engineers: Ethics and Decision Making in Biomedical and Biosystem Engineering. Academic Press. p. 73. ISBN 978-0-08-047610-0.
  7. U.S. Army Corps of Engineers St. Louis District (4 April 2012). Archives Search Report Findings for Field Testing of 2,4,5-T and Other Herbicides (PDF) (Report). p. 116. Retrieved August 8, 2013.CS1 maint: uses authors parameter (link)
  8. Corcoran, Charles A. (December 1968). "Operational Evaluation of Super-Orange (U)- unclassified". Military Assistance Command Vietnam(MAC-V) to Joint Chief of Staff (JCS) message for CINCPAC, USARPAC Ofc Science Adviser. via National Security Archives at George Washington University. Missing or empty |url= (help)
  9. DGSC-PI Memorandum for the record: Herbicides reformulation thereof (Operation Guns and Butter meeting) (Report). Dow Chemical Company. September 9, 1966.
  10. "Herbicides" entry in Spencer C. Tucker, ed. (2011). The Encyclopedia of the Vietnam War (2nd ed.). ABC-CLIO. ISBN 978-1-85109-961-0.
  11. Wright, Laura. "New Study Finds Agent Orange Use Was Underestimated". Scientific American, April 17, 2003.
  12. "Herbicide Tests and Storage in the U.S." Department of Veterans Affairs. Retrieved 16 June 2010.
  13. "Herbicide Tests and Storage Outside the U.S." Department of Veterans Affairs. Retrieved 16 June 2010.
  14. Fryer, J. D.; Blackman, G. E. (January 1972). "Preliminary Proposals for the Study of Persistence of Herbicides in Forest and Mangrove Soil". NAS committee on the effect of Herbicides in Vietnam. National Academy of Science, (NAS). Missing or empty |url= (help)
  15. Pelletier, Richard. "Agent Purple Deadlier than Agent Orange." Bangor Daily News, 1 July 2005.

Further reading
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